4 Unconventional Things You Didn’t Know Were Making You Fat

The human body is a strange and wondrous place. There are many reasons why a person might gain weight. Eating the wrong kind of food, the wrong amount of food, or not enough exercise are well established reasons. However, there are some no-so-common reasons that might play a role as well.

Road Traffic Noise

Exposure to a combination of road traffic, rail, and aircraft noise may pose the greatest risk of acquiring a spare tire — otherwise known as central obesity, and thought to be one of the most harmful types of fat deposition around the body. In a recent study researchers assessed how much road traffic, rail, and aircraft noise 5075 people living in five suburban and rural areas around Stockholm, Sweden, had been exposed to since 1999. The analysis indicated no link between road traffic noise and BMI. But there was an association between road traffic noise and waist size, with a 0.21 cm increase for every additional 5 dB increase in exposure, although this was only significant among women. Similarly, there was a link to waist:hip ratio, with a change of 0.16 for every 5 dB increase in noise exposure to road traffic; this association was stronger in men.

What gives? Noise exposure may be an important physiological stressor and bump up the production of the hormone cortisol, high levels of which are thought to have a role in fat deposition around the middle of the body, researchers suggest. Additionally, noise exposure might disrupt sleep, another known factor to contribute to weight gain.

Too Many Food Choices

A new study in mice by researchers in the Virginia Tech College of Agriculture and Life Sciences and the Edward Via College of Osteopathic Medicine has shown that the environment in which a child lives may be an equal if not stronger force in determining obesity than their mother’s diet. Researchers found that having too many food choices increases the obesity problem. In fact, researchers found that having a choice of a high-fat and low-fat diet does not help — offspring in this situation tended to eat even more. Their findings were recently released in the journal Endocrinology. “We like variety,” said Deborah Good, an author of the paper and an associate professor of human nutrition, foods, and exercise at Virginia Tech. “But when there is a choice, we eat more than when there is not any variety.” Moral of the story? Simple is better for our waistlines.

The Inability To Stay Warm

A new study suggests that a biological inability to create sufficient core body heat could be linked to the obesity epidemic. The study found that obesity is associated with a significant reduction of body core temperature during daytime hours. Journal Editor Francesco Portaluppi explains that the reduced ability of obese people to spend energy as heat compared to lean individuals could result in long term weight gain (about 2 kg (4.5 lb.)) per year, depending on the lifestyle. “Since body core temperature represents a marker of energy expenditure, results from this study suggest that a diurnal thermogenic handicap can play a crucial role in favoring weight gain in obese subjects,” said article co-author Pietro Cortelli, MD, Ph.D. The fix? Generate more body heat. And how does one do that? Exercise of course!

Environmental Pollution

A team of Spanish scientists, which includes several researchers from the University of Granada, has confirmed that there is a relation between the levels of certain environmental pollutants that a person accumulates in his or her body and their level of obesity. Subjects with more pollutants in their bodies tend to have higher levels of cholesterol and triglycerides, which are important risk factors for cardiovascular disease. The research has analyzed the levels of pollutants accumulated in adipose tissue (fat) in nearly 300 men and women, who were attended in the surgery services of two hospitals in the province of Granada (Spain). The substances analyzed, known as persistent organic pollutants (POPs), can remain in the environment for years, even decades, without degrading.

“Humans are exposed to POPs mainly through diet. Besides, POPs accumulate gradually in body fat, and this is the reason why the median levels in our study give us an idea of an individual’s accumulated exposition over a number of years,” says Juan Pedro Arrebola, the main author of the article. There is evidence that human exposure to certain chemical substances called “obesogenic” could favor the growth and proliferation of adipocytes (fat cells), and provoke therefore an increase in body fat.

Do your best to avoid exposing yourself unnecessarily to chemicals that might be problematic. Perhaps the most powerful way to do this is avoid processed food which is loaded with these chemicals from their own packaging.

Recommendation for vitamin D intake was miscalculated, is far too low, experts say

Researchers at UC San Diego and Creighton University have challenged the intake of vitamin D recommended by the National Academy of Sciences (NAS) Institute of Medicine (IOM), stating that their Recommended Dietary Allowance (RDA) for vitamin D underestimates the need by a factor of ten.

In a letter published last week in the journal Nutrients the scientists confirmed a calculation error noted by other investigators, by using a data set from a different population. Dr. Cedric F. Garland, Dr.P.H., adjunct professor at UC San Diego’s Department of Family Medicine and Public Health said his group was able to confirm findings published by Dr. Paul Veugelers from the University of Alberta School of Public Health that were reported last October in the same journal.

“Both these studies suggest that the IOM underestimated the requirement substantially,” said Garland. “The error has broad implications for public health regarding disease prevention and achieving the stated goal of ensuring that the whole population has enough vitamin D to maintain bone health.”

The recommended intake of vitamin D specified by the IOM is 600 IU/day through age 70 years, and 800 IU/day for older ages. “Calculations by us and other researchers have shown that these doses are only about one-tenth those needed to cut incidence of diseases related to vitamin D deficiency,” Garland explained.

Robert Heaney, M.D., of Creighton University wrote: “We call for the NAS-IOM and all public health authorities concerned with transmitting accurate nutritional information to the public to designate, as the RDA, a value of approximately 7,000 IU/day from all sources.”

“This intake is well below the upper level intake specified by IOM as safe for teens and adults, 10,000 IU/day,” Garland said. Other authors were C. Baggerly and C. French, of GrassrootsHealth, a voluntary organization in San Diego CA, and E.D. Gorham, Ph.D., of UC San Diego.

via Recommendation for vitamin D intake was miscalculated, is far too low, experts say — ScienceDaily.

Dr. Court’s Commentary – 

I’ve been recommending anywhere from 4,000-6,000IU/day of vitamin D (supplementally) to my patients for many years. This is consistent with the above recommendations that people get about 7,000IU/day from all sources. Vitamin D deficiency is a culprit in many diseases of aging, and the IOM’s recommendations were far too low. It’s good to see this becoming more well recognized.

Only one study has been done (that I can find) that actually measured how much vitamin D human beings use on a daily basis. The conclusion of that study was that humans use about 4,000-6,000IU per day (hence my recommendation). In that context, the IOM’s recommendation of 600-800IU/day becomes even more startling.

Do Antibiotics Raise Diabetes Risk via Gut Microbiota?

People who take multiple courses of antibiotics may face an increased risk of developing both type 1 and type 2 diabetes, potentially through alterations in gut microbiota, conclude US researchers.

The team, led by Ben Boursi, MD, a postdoctoral researcher in the department of gastroenterology at the University of Pennsylvania, Philadelphia, found that the risk of diabetes was increased by up to 37%, depending on the type of antibiotic and the number of courses prescribed.

“Overprescription of antibiotics is already a problem around the world as bacteria become increasingly resistant to their effects,” commented Dr Boursi in a statement.

“Our findings are important, not only for understanding how diabetes may develop, but as a warning to reduce unnecessary antibiotic treatments that might do more harm than good.”

The study was published online ahead of print March 24 in the European Journal of Endocrinology.

The More Courses of Antibiotics, the Greater the Risk

Dr Boursi explained that studies both in animal models and humans have shown an association between changes in gut microbiota in response to antibiotic exposure and obesity, insulin resistance, and diabetes.

Speaking to Medscape Medical News, he noted: “In mice, we know that germ-free mice are lean and, by fecal transplantation, we can transmit obesity to them. We also know that low dose of penicillin may induce obesity in mice models.”

He added that there have been several studies in humans indicating that exposure to antibiotics in early childhood is associated with an increased risk of obesity in later life, while other investigations have reported differences in gut microbiota between people with and without diabetes.

To investigate further, Dr Boursi and colleagues conducted a nested case-control study using data from the Health Improvement Network (THIN), a UK population-based database, from which they identified 1,804,170 patients with acceptable medical records.

As diabetes is associated with an increased risk of infection, the team wanted to exclude all cases with prediabetes or undiagnosed diabetes. To do that, they removed all patients diagnosed with diabetes within 183 days of starting follow-up and included only patients with exposure to antibiotics more than 1 year prior to the index date.

From the original cohort, they were able to select 208,002 diabetes patients and 815,576 controls matched for age, sex, general practice site, and duration of follow-up before the index date.

Conditional logistic regression analysis revealed that exposure to a single antibiotic prescription was not associated with an increased risk of diabetes, adjusted for body mass index (BMI), smoking, last blood glucose level, and the number of infections before the index date, alongside a history of coronary artery disease and hyperlipidemia.

However, treatment with two to five courses of antibiotics was linked to an increased risk of diabetes with penicillin, cephalosporins, macrolides, and quinolones, at adjusted odds ratios (ORs) ranging from 1.08 for penicillin to 1.15 for quinolones.

The highest risk for diabetes was seen among people who received more than five courses of quinolones, at an adjusted OR of 1.37. An increased risk of diabetes was also seen in patients who took more than five courses of tetracyclines, at an adjusted OR of 1.21.

Interestingly, the researchers were unable to find an association between diabetes risk and treatment with imidazole, antiviral drugs, and antifungals, regardless of the number of courses.

To account for further possible confounding factors, the researchers repeated the analysis only in individuals without skin or urinary-tract infections, which are more common among diabetes patients. This had no impact on the results.

Next Steps

When the analysis was restricted to type 1 diabetes, the risk was increased only following exposure to more than five courses of penicillin or two to five courses of cephalosporin, at odds ratios of 1.41 and 1.63, respectively.

Commenting on the findings, study coauthor Yu-Xiao Yang, MD, assistant professor of medicine and epidemiology, University of Pennsylvania, pointed out their investigation was observational in nature.

“We are not able to establish cause and effect necessarily, but it is actually pretty consistent with the experimental data, which is more definitive in terms of the animal data than in humans.”

Dr Yang said that the next step for the team will be to expand their focus, as the antibiotics data “provide indirect evidence suggesting the importance of gut microbiota on metabolic outcomes, including diabetes.”

Describing their findings as “important evidence,” he concluded: “Based on this indirect evidence and existing data in animals, we are planning to more directly investigate the effect of altered microbe environments in humans.”

The work was supported by the National Center for Research Resources and the National Center for Advancing Translational Sciences at the National Institutes of Health. The authors have reported no relevant financial relationships.

Eur J Endocrinol. 2015. Published online March 24, 2015. Abstract

via Do Antibiotics Raise Diabetes Risk via Gut Microbiota?.

High Fructose Corn Syrup Will Kill You!

A common additive used in a wide range of commercially available processed foods such as soft drinks, salad dressings, cookies and cakes, breads and breakfast cereals has been poisoning people for several decades now. In fact, research studies have shown that it causes metabolic syndrome which in turn contributes a great deal to an increase in body weight and incidence of degenerative diseases such as diabetes, heart disease, Alzheimer’s and fatty liver. This additive is high-fructose corn syrup (HFCS).

Sucrose, or regular table sugar, consists of 50 percent fructose and 50 percent glucose, whereas HFCS can contain up to 90 percent fructose, almost twice the fructose of common table sugar. In terms of calories, both HFCS and table sugar provide 4 calories per gram, but the issue is the metabolism of excessive amounts of fructose.

High-Fructose Corn Syrup History

In the ninth century, the Japanese invented a sweetener derived from starches. This syrup is being used even today as “traditional sweetener.” In the 17th century, Andreas Sigismund Marggraf, a German chemist, discovered another starch-based sweetener called glucose.

During the times of Napoleonic wars, blockades on sugarcane imports from the West Indies encouraged laboratories to work on development of alternative sweeteners. Dextose or D-glucose was developed in 1801, and in 1811 a Russian scientist created glucose by overcooking potato starch in sulfuric acid.

No further progress was made till the 1950s when the Japanese invented the HFCS. In America, cane sugar continued to be used as the sweetener of choice until the 1970s prior to the introduction of the less expensive sweeteners such as maltodextrin and HFCS that were derived from corn. HFCS was introduced to the food industry after the developmental process was perfected.

Why is HFCS so popular with the food industry?

First and foremost, its sweetness is comparable to that of table sugar. Secondly, it maintains the quality of condiments and drinks for a longer period. Third, it provides a soft, moist texture to baked food such as snack bars and cookies by retaining moisture and resisting crystallization after baking. It is HFCS that gives baked foods flavor and superior browning quality. The sugars present in HFCS get fermented quickly and easily, making it possible to produce sweeter bread. Fourth, it is much cheaper compared to table sugar. Lastly, it is easily added to just about anything. It is generally produced in a liquid form making its incorporation into food and drink products a very simple task for automated equipment that is so common in food production today.

Health Effects of HFCS

The statistics released by the Department of Agriculture in the U.S. show that the average consumption of HFCS has increased from 0.5 pounds per person per year to 60 pounds per person per year over the past four decades.

During the same period, there has been a threefold increase in obesity rates and a seven fold increase in the incidence of diabetes. HFCS may not be singularly responsible for this, but its effect cannot be ignored.

A number of short-term clinical studies have shown that ingesting sweetened beverages is not good for health. Results of one study showed that people gained weight and experienced an increase in blood pressure and inflammatory markers. In another study, scientists observed an increase in visceral fat and triglyceride levels and stimulation of lipogenesis (producing more fat). In yet another trial wherein the effects of water, milk, diet cola and sugar-sweetened cola were compared, the results showed that sugar-sweetened beverage contributed to an increase in liver and visceral fat and elevated triglyceride levels when consumed for 6 months at the rate of just 16 ounce per day (i.e about one soda).

Fructose also increases gut permeability allowing potent bacterial toxins out of the gut and into surrounding immune tissue. This generates an inflammatory response. If this response continues for long periods of time, systemic levels of inflammation begin to rise increasing the risk of obesity, cancer, heart disease, diabetes, Alzheimer’s disease, osteoporosis, arthritis and more. And most studies have shown it only takes the fructose present in one soda to produce these adverse effects.

You may use sugar in any form, but it definitely causes obesity as well as other diseases when consumed in pharmacologic doses. HFCS is often a marker of nutrient-deficient, poor quality and disease-causing industrial food products. It should be severely limited in your diet.

Corn Industry’s Marketing Push

The corn processing industry thrives on doubt and confusion. The Corn Refiners Association skillfully uses the print and television media for massive advertising campaigns in order to dispel the fear among people. The industry also asserts through medical doctors’ as well as nutrition experts’ opinion that HFCS is same as cane sugar and it is a “natural” product if used in moderation. They do this for nothing but commercial benefit though they themselves are aware that this is not true. No independent medical or nutrition experts recommend the consumption of HFCS. My experience lecturing at the 2012 Food and Nutrition Convention and Expo was enlightening. The Corn Refiners Association sponsored lecture after lecture on HFCS and how it was not the villain it was made out to be. In fact, they asserted it could be consumed as part of a healthy diet. They also skillfully shifted the focus from HFCS to the epidemic of inactivity in the US, blaming it instead of processed food for the skyrocketing chronic disease rates in this country.

Do yourself a favor and avoid HFCS. While the corn and sugar industry continues to deceive, they are killing thousands of Americans to make a dollar. It rings eerily similar to Big Tobacco of the 50s and 60s. You don’t want to find out too late that money and corporate interest mislead you into believing it was safe when the research is clear now.

8 Pictures That Perfectly Describe Leaky Gut (and how to fix it)!

Our gut has many functions. Changes in the balance of bacteria, leaky gut, bacterial overgrowth and other factors may affect how your GI system works. This may have systemic consequences.

Increasingly, chronic disease is being seen as stemming from altered gut function. Follow the arrows to see what the risks for chronic disease are. All of the factors that increase chronic disease risk adversely affect gut function too.

The bacteria in our gut is EXTREMELY important and there is lots of it. If it becomes unbalanced, all symbiotic functions are lost.

These disease have all been linked to changes in the balance of bacteria in the gut

The gut forms an important barrier between what’s inside the intestines and what gets absorbed. Those blue structures highlighted by the orange arrows are tight junctions. They hold the cells together do nothing can squeeze between them and pass into the local blood supply unchecked. This is a very important function.

All of these factors can lead to breakdown of the tight junctions and leaky gut. NSAIDs are pain relievers like Aspirin, Aleve, Advil, etc. SIBO is an acronym for small intestinal bacterial overgrowth. Additionally, low exercise levels is a stressor under the category of physical stress. 

LPS are toxic structures located on the surface of the bacteria in the gut. When bacteria die, they are free to potentially enter our bloodstream. This would happen through a leaky gut. If this occurs your body’s response is inflammatory. This inflammation alters your energy levels, your mood, and eventually increases the risk of heart disease, cancer, diabetes, and many other chronic diseases.

Start in the upper left corner of the diagram and follow the arrows to see how a leaky gut leads to disease.

Fixing the problem – follow these steps to fix your leaky gut

1. Get advanced stool testing done to properly evaluate your GI health.
2. Eat a paleo-inspired diet full of vegetables, healthy proteins, and healthy fats.
3. Avoid food sensitivities.
4. Consume fermented foods regularly.
5. Supplement according to your needs. This might include nutrient repletion, probiotics, whey protein, anti-inflammatory herbs like curcumin, or fish oil.
6. Exercise vigorously 3-4 times per week.
7. Eliminate artificial sweeteners, do not overuse NSAIDs, antibiotics, or alcohol, manage your stress, and manage your stress.

If you do all of that, your can heal your leaky gut and feel better than you ever thought you could!

A new take on artificial sweeteners

Artificial sweeteners are supposed to be good for you. Go ahead, ask your doctor or a dietitian. They’ll tell you they are a great way to get your sweet fix without any consequences. When compared to full sugar soda they’re supposed lower your caloric intake, they reduce your risk of being overweight, they reduce your risk of diabetes, they reduce your risk of heart disease and so on and so forth. Unfortunately, that’s not what the recent research is showing.

Non-caloric artificial sweeteners are among the most widely used food additives worldwide, regularly consumed by lean and obese individuals alike. They were introduced over a century ago as means for providing sweet taste to foods without the associated high energy content of caloric sugars, yet supporting scientific data on safety and efficacy remain sparse and controversial. While some data has shown they boost blood sugar very little, other evidence has linked them to type 2 diabetes and weight gain. These are the conditions they were created to prevent. The question then is how do artificial sweeteners create physiologic change capable of making us unhealthy? A new study is providing some input.

Most artificial sweeteners pass through the human gastrointestinal tract without being digested by the person consuming them, thus directly encountering the intestinal microbiota (bacteria), which plays central roles in regulating multiple physiological processes. These artificial sweeteners alter the balance of the bacteria present in our gastrointestinal tract, thus adversely affecting many of these important processes.

This new study titled Artificial Sweeteners Induce Glucose Intolerance by Altering the Gut Microbiota, has demonstrated that consumption of commonly used artificial sweeteners drives the development of glucose intolerance (high blood sugar) through changes in the composition of the intestinal microbiota. Further, the use of antibiotics eliminates these effects. This confirms the bacteria play a central role in the metabolic changes. Now, I’m not saying we should all be on antibiotics. This study used antibiotics to confirm the theory that artificial sweeteners adversely affects physiology through changes in the intestinal microbiome. This is not a viable option in real life as this can have severe consequences long term.

The exact mechanism through which these adverse physiologic changes occur is not completely understood, but it appears to be related to a change in the composition certain types of bacteria. It creates a problem known as dysbiosis (unbalanced growth of bacteria). This dysbiosis results in the same bacterial profile known to be associated with diabetes, obesity, and over-extraction of calories from food.

So, artificial sweeteners create the same problem they are intended to prevent? Yes. So what do you drink, you say? Water. Water is the perfect hydrating liquid. If you are very physically active, a rehydrating drink with electrolytes and some carbohydrate replacement is fine during an intense workout. If you want to have something sweet, have a real soda. But do it only VERY infrequently!

Want to know more? Sign up for our FREE WEBINAR called “Chronic Disease Hates Your Guts!” November 11th at 7PM. We’ll discuss the importance of a healthy gastrointestinal systems as it relates to the most common complaints in medical practice. You don’t want to miss this!

Register here: 

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Why Your Pants Shouldn’t Fit!

Have you heard about a metric used by health professionals called the waist to hip ratio? It’s a measure of abdominal obesity. Abdominal obesity is strongly associated with an increased risk of type 2 diabetes, cardiovascular disease and death, even after controlling for other factors like overall weight. In the waist to hip ratio, waist circumference and waist size are compared to hip size. Several organizations have defined cut points for abdominal obesity with different cut points for men and women. 

According to the World Health Organization (WHO) the waist circumference should be measured at the midpoint between the lower margin of the last palpable rib and the top of the iliac crest, using a stretch‐resistant tape that provides a constant 100 g tension. Hip circumference should be measured around the widest portion of the buttocks, with the tape parallel to the floor. Practically, however, the waist is more conveniently measured simply at the smallest circumference of the natural waist, usually just above the belly button, and the hip circumference may likewise be measured at its widest part of the buttocks or hip. The WHO states that abdominal obesity is defined as a waist–hip ratio above 0.90 for males and above 0.86 for females. In layman’s terms, if your belly is bigger than your butt, you’ve got a problem.

I recently measured my waist to hip ratio. It was about 0.83 – well within the healthy range. Here’s how I found it. My waist measures about 34 inches. My hips measure about 41 inches. If you divide 34 by 41 you get 0.82926 repeating or about 0.83. For reference I’m about 5’11”, 187 pounds and about 18% body fat.

So what the heck does this have to do with my pants?

Everything! It’s a quick, down and dirty way to assess your waist to hip ratio. When I buy pants, the waist NEVER fits. It’s ALWAYS too big. This is a good thing. The reason it’s too big is I have to be able to fit them over my legs and butt. If you talk to anyone who works out or is athletic, they’ll tell you the same thing. If a pair of pants fits on the waist, it’s super tight in the legs and butt. If it fits the butt and legs, the waist is enormous. It’s a good, if not maddening, problem to have. Here’s what I mean:

Just about all of my pants are 1-3 inches too big in the waist, by they fit everywhere else!

This is what you don’t want:

This man’s waist is clearly larger than his hips.

Everyone should have an idea of what their waist to hip ratio is. It’s a simple and easy metric for assessing your general health. Don’t want to take the time to measure it? Fine, how do your pants fit? Are the waists too big but legs and butt just fine? GREAT. Are you having trouble finding pants to fit your waist because when they fit your waist they fall off the rest of you? Uh-oh. Time to make some lifestyle changes and get healthy!

Sugar, Sugar, Sugar

Do you eat “healthy?” Or, perhaps a better question should be ‘DID you think you ate healthy,’ before this post?

Food labeling is VERY misleading. Don’t be fooled. Get educated! High sugar consumption increases the risk of diabetes, heart disease, high blood pressure and Alzheimer’s disease!

Is this really a “healthy” diet?

 

 

7 Simple Steps to a Sound Mind, Body, and Soul

These days it seems everyone is looking for the quick fix, the pill, the next fad diet that will change their lives. They are looking to increase energy, feel younger, be stronger or live to 100. I’m here to tell you there’s no quick fix. Surprised? I didn’t think so.

Most people are aware there is no such thing as a quick fix, however, there are things you can do for yourself that will help you live a long, happy life. And they’re simple!

Step 1 – Eat a healthy diet

This seems like a no brainer, but often people neglect this one. We take better care of our cars than we do our bodies yet it is known that most chronic diseases are diet related.

Step 2 – Exercise

Another no brainer, yet the average American spends 35 hours a week watching TV and just two hours a week exercising. Low physical activity is linked to many chronic diseases including Alzheimer’s disease, heart disease and diabetes.

Step 3 – Balance brain function

A phenomenon called hemisphericity can change the way the hemispheres of your brain communicate with each other. This can lead to foggy thinking, processing problems, attention issues, headaches and much more. A functional neurologist can find out if this is affecting you.

Step 4 – Balance brain chemistry

This goes along with the above phenomenon. Neurotransmitters are necessary for proper brain function. We must have the correct balance of these neurotransmitters because too little of one or too much of another can cause symptoms like depression, anxiety, migraines, attention issues, or even brain degeneration. Everything from genetics to diet can influence the balance of neurotransmitters in your brain.

Step 5 – Reduce inflammation

Chronic inflammation is silent and often symptom free. It destroys tissues, causes atherosclerosis, heart disease, cancer, Alzheimer’s disease and diabetes. It also accelerates aging. Chronic inflammation is often diet related and is increasingly being seen as the cause of most diseases that affect humans.

Step 6 – Manage your stress

Research is very clear that stress increases levels of inflammation in the body through a process called glucocorticoid receptor resistance. Essentially, with high levels of stress the body over produces cortisol. With this huge supply of cortisol, tissues begin to ignore it and compensate by producing high levels of inflammatory molecules. Managing stress can involve many things including exercise, meditation, and cognitive behavioral therapy to name a few.

Step 7 – Stay positive

Interesting research has concluded that pessimism is associated with poorer health outcomes in the general population. Negative mood states are associated with increased activation of the fight or flight system (sympathetic nervous system) and decreased activation of the rest and digest system (parasympathetic nervous system). Again, this leads to an increase in systemic inflammation!

Want more information? Join Dr. Vreeland for a FREE webinar next week on June 5th from 7-8pm. We’ll discuss all of the above in detail and tell you how you can live happy and healthy! We look forward to seeing you there!

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The Best Weight Loss Diet Is?

New information confirms what I have been telling people for years.  A low fat diet is bad for your health and a low glycemic diet is the best diet for weight loss.  The recent study was published in the Journal of the American Medical Association.  For more information, watch the short video below.